<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0120-5633</journal-id>
<journal-title><![CDATA[Revista Colombiana de Cardiología]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. Colomb. Cardiol.]]></abbrev-journal-title>
<issn>0120-5633</issn>
<publisher>
<publisher-name><![CDATA[Sociedad Colombiana de Cardiologia. Oficina de Publicaciones]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0120-56332024000100057</article-id>
<article-id pub-id-type="doi">10.24875/rccar.23000055</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Perfil de la hipertensión arterial non-dipper: fisiopatología, etiopatogenia y relación con desenlaces cardiovasculares]]></article-title>
<article-title xml:lang="en"><![CDATA[Non-dipper hypertension: its pathophysiology, etiology and association with cardiovascular outcomes]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Chará]]></surname>
<given-names><![CDATA[Camilo J.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Andrade]]></surname>
<given-names><![CDATA[Andrés F.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pinzón]]></surname>
<given-names><![CDATA[María V.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[López]]></surname>
<given-names><![CDATA[Nelson A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad del Cauca Departamento de Medicina Interna ]]></institution>
<addr-line><![CDATA[Popayán ]]></addr-line>
<country>Colombia</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>02</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>02</month>
<year>2024</year>
</pub-date>
<volume>31</volume>
<numero>1</numero>
<fpage>57</fpage>
<lpage>64</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_arttext&amp;pid=S0120-56332024000100057&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_abstract&amp;pid=S0120-56332024000100057&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_pdf&amp;pid=S0120-56332024000100057&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El comportamiento circadiano de la presión arterial se clasifica en distintos fenotipos que aportan conocimiento adicional sobre el estado mórbido y el pronóstico de los pacientes. La monitorización ambulatoria de la presión arterial (MAPA) es la herramienta ideal para obtener un registro de estas variaciones. Su etiología es multifactorial, incluyendo disregulaciones en el sistema nervioso autónomo, ejes neuroendocrinos y sistema de recambio mineral, mostrando un comportamiento complejo desde el punto de vista fisiopatológico. Existen diversas asociaciones de fenotipos circadianos alterados, como el non dipper, con desenlaces cardiovasculares adversos como mortalidad cardiovascular, enfermedad coronaria, cerebrovascular, enfermedad renal crónica y hospitalización por falla cardiaca. El conocimiento de estos aspectos es fundamental para definir grupos especiales de riesgo en pacientes con hipertensión.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Circadian blood pressure variations are classified into different phenotypes that provide additional knowledge about patients’ morbidity and prognosis of patients. Ambulatory blood pressure monitoring (ABPM) is the ideal tool to obtain a record of these variations. The etiology of non-dipper hypertension is multifactorial, including dysregulations in the autonomic nervous system, neuroendocrine axes, and mineral turnover system, with complex pathophysiological behavior. There are many associations between altered circadian phenotypes (such as the non dipper phenotype) and adverse cardiovascular outcomes such as cardiovascular mortality, coronary and cerebrovascular disease, chronic kidney disease, and heart failure. Knowledge of these aspects is essential to define special risk groups in patients with hypertension.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Presión arterial]]></kwd>
<kwd lng="es"><![CDATA[Hipertensión arterial]]></kwd>
<kwd lng="es"><![CDATA[Non dipping]]></kwd>
<kwd lng="es"><![CDATA[Hipertensión nocturna]]></kwd>
<kwd lng="es"><![CDATA[Fisiopatología]]></kwd>
<kwd lng="es"><![CDATA[Riesgo cardiovascular]]></kwd>
<kwd lng="en"><![CDATA[Blood pressure]]></kwd>
<kwd lng="en"><![CDATA[Arterial hypertension]]></kwd>
<kwd lng="en"><![CDATA[Non-dipping]]></kwd>
<kwd lng="en"><![CDATA[Nocturnal hypertension]]></kwd>
<kwd lng="en"><![CDATA[Pathophysiology]]></kwd>
<kwd lng="en"><![CDATA[Cardiovascular risk]]></kwd>
</kwd-group>
</article-meta>
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