<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0120-8748</journal-id>
<journal-title><![CDATA[Acta Neurológica Colombiana]]></journal-title>
<abbrev-journal-title><![CDATA[Acta Neurol Colomb.]]></abbrev-journal-title>
<issn>0120-8748</issn>
<publisher>
<publisher-name><![CDATA[Asociación Colombiana de Neurología]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0120-87482024000100008</article-id>
<article-id pub-id-type="doi">10.22379/anc.v40i1.1209</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Síndrome de la arteria de Percheron, a propósito de un caso]]></article-title>
<article-title xml:lang="en"><![CDATA[Percheron artery syndrome: A case report]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Guevara Rodríguez]]></surname>
<given-names><![CDATA[Marbelys]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Bosch Rodríguez]]></surname>
<given-names><![CDATA[Bettsy Bell]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Medina Hernández]]></surname>
<given-names><![CDATA[Larry]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Bordón Hernández]]></surname>
<given-names><![CDATA[Mercedes]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Luna Capote]]></surname>
<given-names><![CDATA[Amalia Inés]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Ciencias Médicas de Villa Clara  ]]></institution>
<addr-line><![CDATA[Santa Clara ]]></addr-line>
<country>Cuba</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Hospital Provincial Clínico Quirúrgico Universitario Arnaldo Milián Castro  ]]></institution>
<addr-line><![CDATA[Santa Clara ]]></addr-line>
<country>Cuba</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>03</month>
<year>2024</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>03</month>
<year>2024</year>
</pub-date>
<volume>40</volume>
<numero>1</numero>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_arttext&amp;pid=S0120-87482024000100008&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_abstract&amp;pid=S0120-87482024000100008&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_pdf&amp;pid=S0120-87482024000100008&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Introducción: El síndrome de Percheron es una entidad infrecuente de difícil determinación clínica y que es causada por la oclusión de la arteria de Percheron.  Presentación del caso: Paciente masculino de 71 años que llega obnubilado al servicio de urgencias, con disartria, hemiparesia izquierda, signo de Babinski bilateral, ptosis palpebral derecha, con exotropía, limitación total de la aducción del ojo derecho y abducción parcial del izquierdo, con imposibilidad de supraducción e infraducción de ambos ojos. Además, tenía reflejo oculocefálico vertical ausente bilateralmente y horizontal derecho ausente, pupilas anisocóricas y reflejo fotomotor ausente en ambos ojos. La imágenes obtenidas por resonancia magnética nuclear (RMN) (T1W, T2, T2-FLAIR y técnicas de difusión) demostraron que hubo un infarto de la arteria de Percheron. El paciente se mantuvo por 10 días hospitalizado y evolucionó favorablemente.  Discusión: El síndrome de Percheron cursa con una triada clásica caracterizada por alteración de la conciencia, deterioro cognitivo y parálisis de la mirada vertical, a lo que se le añade hemiplejía, ataxia cerebelosa y déficit oculomotor y, si se acompaña de lesiones mesencefálicas, se produce un síndrome tálamo-peduncular. La sospecha clínica de esta patología constituye un desafío en la práctica médica, debido a la infrecuencia de presentación, variabilidad de alteraciones neurológicas y la dificultad que ofrecen los estudios imagenológicos de urgencia.  Conclusiones: Hemos descrito una entidad infrecuente y de difícil determinación clínica, en la cual, los criterios para el diagnóstico son fundamentalmente imagenológicos, siendo de elección la RMN. La evolución y el pronóstico dependerán de la extensión del proceso, las estructuras involucradas, el diagnóstico y su tratamiento oportuno.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Introduction: Percheron Syndrome is an infrequent entity that is difficult to determine clinically, caused by the occlusion of the Percheron artery.  Case presentation: We present a 71-year-old male patient admitted to the emergency department: obtunded, with dysarthria, left hemiparesis, bilateral Babinsky's sign, right palpebral ptosis, with exotropia, total adduction limitation of the right eye and partial adduction of the right eye. abduction of the left with impossibility of supraduction and infraduction of both eyes. Absent vertical oculocephalic reflex bilaterally and absent right horizontal. Anisochoric pupils, photomotor reflex absent in both eyes. The images obtained by Nuclear Magnetic Resonance (T1W, T2, T2-FLAIR and diffusion techniques) showed infarction of the Percheron artery. He remained hospitalized for 10 days, and evolved favorably.  Discussion. Percheron Syndrome presents with a classic triad characterized by altered consciousness, cognitive impairment and paralysis of vertical gaze, to which is added hemiplegia, cerebellar ataxia and oculomotor deficit, if accompanied by mesencephalic lesions, producing a Thalamopeduncular Syndrome. The clinical suspicion of this pathology constitutes a challenge in medical practice, due to the infrequency of presentation, variability of neurological alterations and the difficulty offered by emergency imaging studies.  Conclusions. We have described a rare entity that is difficult to determine clinically, in which the criteria for diagnosis are fundamentally imaging, with MRI being the choice. The evolution and prognosis will depend on the extent of the process, structures involved in addition to the diagnosis and timely treatment.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[alteración de la conciencia]]></kwd>
<kwd lng="es"><![CDATA[deterioro cognitivo]]></kwd>
<kwd lng="es"><![CDATA[parálisis de la mirada vertical]]></kwd>
<kwd lng="es"><![CDATA[resonancia magnética nuclear]]></kwd>
<kwd lng="es"><![CDATA[síndrome de Percheron]]></kwd>
<kwd lng="es"><![CDATA[síndrome tálamopeduncular]]></kwd>
<kwd lng="en"><![CDATA[Altered consciousness]]></kwd>
<kwd lng="en"><![CDATA[Cognitive impairment]]></kwd>
<kwd lng="en"><![CDATA[Vertical gaze paralysis]]></kwd>
<kwd lng="en"><![CDATA[Nuclear magnetic resonance]]></kwd>
<kwd lng="en"><![CDATA[Percheron syndrome]]></kwd>
<kwd lng="en"><![CDATA[Thalamopeduncular syndrome]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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