<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0121-0793</journal-id>
<journal-title><![CDATA[Iatreia]]></journal-title>
<abbrev-journal-title><![CDATA[Iatreia]]></abbrev-journal-title>
<issn>0121-0793</issn>
<publisher>
<publisher-name><![CDATA[Universidad de Antioquia]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0121-07932017000400423</article-id>
<article-id pub-id-type="doi">10.17533/udea.iatreia.v30n4a06</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Inflamación y respuesta inmune innata: participación de las lipoproteínas de alta densidad]]></article-title>
<article-title xml:lang="en"><![CDATA[Inflammation and innate immune response: role of high-density lipoprotein]]></article-title>
<article-title xml:lang="pt"><![CDATA[Inflamação e resposta imune inata: participação das lipoproteínas de altadensidade]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Marín-Palma]]></surname>
<given-names><![CDATA[Damariz]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Taborda]]></surname>
<given-names><![CDATA[Natalia Andrea]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Urcuqui-Inchima]]></surname>
<given-names><![CDATA[Silvio]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hernandez]]></surname>
<given-names><![CDATA[Juan Carlos]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Antioquia  ]]></institution>
<addr-line><![CDATA[Medellín ]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad de Antioquia  ]]></institution>
<addr-line><![CDATA[Medellín ]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad de Antioquia  ]]></institution>
<addr-line><![CDATA[Medellín ]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Universidad de Antioquia  ]]></institution>
<addr-line><![CDATA[Medellín ]]></addr-line>
<country>Colombia</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2017</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2017</year>
</pub-date>
<volume>30</volume>
<numero>4</numero>
<fpage>423</fpage>
<lpage>435</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_arttext&amp;pid=S0121-07932017000400423&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_abstract&amp;pid=S0121-07932017000400423&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_pdf&amp;pid=S0121-07932017000400423&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN Las lipoproteínas de alta densidad (HDL) tienen como función transportar el exceso de colesterol desde los tejidos hacia el hígado, para ser excretado; y de este modo contribuyen al control de las enfermedades cardiovasculares. Además, las HDL pueden modular la respuesta inmune, por sus propiedades anti-inflamatorias, antioxidantes y anti-apoptóticas, entre otras. A nivel celular, las HDL pueden modificar las balsas lipídicas, las cuales son determinantes en la activación de la respuesta inmune frente a patógenos o agentes extraños. En algunas patologías como la sepsis, las HDL participan mediando la eliminación del lipopolisacárido (LPS) a través de su captura y posterior eliminación en el hígado; esto conlleva a una modulación negativa de la expresión del TLR4, principal receptor del LPS. También se ha reportado que las HDL modulan la respuesta inflamatoria a través de la regulación de la activación de la cascada del complemento y la expresión de pentraxina 3. Finalmente, la función y los niveles de las HDL se han encontrado particularmente alterados en algunas patologías infecciosas, ateroesclerosis y sepsis, lo que se ha asociado con el progreso o la severidad de la enfermedad.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[SUMMARY The main function of high-density lipoproteins (HDL) is to transport the excess of cholesterol from tissues to the liver, where it is excreted, thus decreasing the risk of atherosclerotic plaques development and cardiovascular diseases establishment. Besides, HDL participates in different processes of the immune response, as its components have anti-inflammatory, anti-oxidative and antiapoptotic activities, among others. At cellular level, these lipoproteins have the ability to modify lipid rafts, critical micro-domains that participate in signaling pathways in response to pathogens. Likewise, it has been reported that HDL have a great impact in sepsis, as they eliminate the excess of LPS in blood and transport it to the liver for its elimination, and downregulate the expression of TLR4, main receptor of this molecule. Also, HDL can modulate humoral innate immune responses through regulating the activation of the complement pathway and the expression of pentraxin 3, resulting in the modulation of inflammatory processes. In fact, it has been reported that in several infectious diseases, atherosclerosis and sepsis, the level and function of HDL are altered, which associates with the progress of the diseases.]]></p></abstract>
<abstract abstract-type="short" xml:lang="pt"><p><![CDATA[RESUMO As lipoproteínas de alta densidade (HDL) têm a fun- ção de transportar o excesso de colesterol desde os tecidos até ofígado para aqui ser excretado e dessa forma contribuem no controlo das doenças cardiovasculares. Além disso, as HDL podemmodular a resposta imune devido as suas propriedades anti-inflamatórias, antioxidantes e antiapopticas, entreoutras. Anívelcelular, asHDLpodemmodificar os rafts lipídicos, que são essências para a ativa- ção da resposta imune contra patógenos ouagentes estranhos. Nalgumas patologias como a septicemia, as HDL participam mediando a eliminação do lipopolissacarídeo (LPS) através da sua captura e posterior eliminação no fígado. Isto leva a uma modulação negativa da expressão de TLR4, orecetor principal do LPS. Também foi reportado que as HDL modulam a resposta inflamatória através da regulação da ativaçãoda cascata do sistema do complemento e a expressão de pentraxina 3. Por último, a função e os níveis das HDL têm sido especialmente encontrados alterados nalgumas doenças infeciosas, em aterosclerose e em septicemia, o qual esta associado aevolução ou a gravidade da doença.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Colesterol]]></kwd>
<kwd lng="es"><![CDATA[HDL]]></kwd>
<kwd lng="es"><![CDATA[Inmunomodulación]]></kwd>
<kwd lng="es"><![CDATA[Respuesta Inmune]]></kwd>
<kwd lng="en"><![CDATA[Cholesterol]]></kwd>
<kwd lng="en"><![CDATA[HDL]]></kwd>
<kwd lng="en"><![CDATA[Immunomodulation]]></kwd>
<kwd lng="en"><![CDATA[Immune Response]]></kwd>
<kwd lng="pt"><![CDATA[Colesterol]]></kwd>
<kwd lng="pt"><![CDATA[HDL]]></kwd>
<kwd lng="pt"><![CDATA[Imunomodulação]]></kwd>
<kwd lng="pt"><![CDATA[Resposta Imune]]></kwd>
</kwd-group>
</article-meta>
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