<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0122-9354</journal-id>
<journal-title><![CDATA[Revista de Medicina Veterinaria]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. Med. Vet.]]></abbrev-journal-title>
<issn>0122-9354</issn>
<publisher>
<publisher-name><![CDATA[Universidad de La Salle]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0122-93542017000300129</article-id>
<article-id pub-id-type="doi">10.19052/mv.4395</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Efecto del clorhidrato de clembuterol en la ganancia de peso y lesiones histológicas en ratones]]></article-title>
<article-title xml:lang="en"><![CDATA[Effect of clenbuterol hydrochloride on weight gain and histological lesions in mice]]></article-title>
<article-title xml:lang="pt"><![CDATA[Efeito do cloridrato de clembuterol no aumento de peso e lesões histológicas em ratos]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Valladares-Carranza]]></surname>
<given-names><![CDATA[Benjamín]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Bañuelos-Valenzuela]]></surname>
<given-names><![CDATA[Rómulo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Peña-Betancourt]]></surname>
<given-names><![CDATA[Silvia D.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Velázquez-Ordóñez]]></surname>
<given-names><![CDATA[Valente]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Echavarría-Cháirez]]></surname>
<given-names><![CDATA[Francisco G.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Ortega-Santana]]></surname>
<given-names><![CDATA[César]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sánchez-Torres]]></surname>
<given-names><![CDATA[Juan E.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Lozano-Carbajal]]></surname>
<given-names><![CDATA[Braulio]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Autónoma de Zacatecas Unidad Académica de Medicina Veterinaria y Zootecnia ]]></institution>
<addr-line><![CDATA[Zacatecas ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Autónoma de Zacatecas  ]]></institution>
<addr-line><![CDATA[Zacatecas ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad Autónoma Metropolitana  ]]></institution>
<addr-line><![CDATA[Xochimilco ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Universidad Autónoma del Estado de México  ]]></institution>
<addr-line><![CDATA[Toluca ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af5">
<institution><![CDATA[,Universidad Autónoma de Zacatecas  ]]></institution>
<addr-line><![CDATA[Zacatecas ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>07</month>
<year>2017</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>07</month>
<year>2017</year>
</pub-date>
<numero>35</numero>
<fpage>129</fpage>
<lpage>136</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_arttext&amp;pid=S0122-93542017000300129&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_abstract&amp;pid=S0122-93542017000300129&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_pdf&amp;pid=S0122-93542017000300129&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El clorhidrato de clembuterol (CCL) es un p-agonista promotor del crecimiento en animales para abasto, pero su uso ilícito ha generado repercusiones en salud pública. Se realizó un modelo biológico con ratones, con el objeto de evaluar el efecto del CCL sobre la ganancia de peso y las lesiones histológicas que ocasiona. Los ratones fueron alimentados con carne de conejo, que previamente fue suplementada con CCL. Treinta y cinco días posexposición se registró el peso corporal; se obtuvo la concentración muscular y sérica de CCL a través de la prueba de ELISA, y se colectaron tejidos (hígado y corazón) para análisis histopatológico. Los valores obtenidos de los animales experimentales (G1 y G2) se analizaron mediante un diseño experimental completamente al azar con dos tratamientos (n = 10), sometidos a un análisis de varianza y comparación de medias con la prueba de Tukey (p &lt; 0,05). Se registró un incremento de peso corporal de 7 g en el G1, contra 4,0 g del G2. El peso del hígado fue de 2,58 g y 1,79, respectivamente (p &lt; 0,05). En el G1 la concentración muscular de CCL fue 5324 pg g-1 y en suero sanguíneo de 4378 pg g-1. Solo se observaron cambios histológicos en tejidos de los ratones del G1. El hígado mostró tumefacción celular, mitosis moderada, picnosis y degeneración hidrópica; en corazón, engrosamiento de fibras, pleomorfismo e hileración nuclear. El CCL favoreció el incremento de peso en los ratones expuestos, y provocó alteraciones estructurales en hígado y corazón.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Clenbuterol hydrochloride (CLB) is a growth-promoting p-agonist in animals for supply, but its illicit use has generated repercussions on public health. A biological model with mice was developed to evaluate the effect of CLB on weight gain and histological lesions. Mice were fed rabbit meat, which was previously supplemented with CLB. Body weight was recorded 35 days post-exposure; muscular and serum concentration of CLB was obtained through the ELISA test, and tissues were collected from liver and heart for histopathological analysis. Values obtained from the experimental animals (G1 and G2) were analyzed by a completely randomized experimental design with two treatments (n = 10), subjected to an analysis of variance and comparison of means with the Tukey test (p&lt;0.05). There was an increase of 7 g in body weight in G1, compared to 4.0 g in G2. Liver weight was 2.58 g and 1.79, respectively (p&lt;0.05). In G1, CLB concentration in muscle was 5324 pg.g-1, and 4378 pg.g-1 in blood serum. Only histological changes were observed in the tissues of Gl mice. Liver showed cellular swelling, moderate mitosis, pyknosis and hydropic degeneration; in addition, fiber thickening, pleomorphism and nuclear atypia were observed in the heart. CLB contributed to weight gain in exposed mice and caused structural alterations in liver and heart.]]></p></abstract>
<abstract abstract-type="short" xml:lang="pt"><p><![CDATA[Resumo O cloridrato de clembuterol (CCL) é um b-agonista promotor do crescimento em animais para abate, porém o seu uso ilícito tem gerado repercussões em saúde pública. Fezse um modelo biológico com ratos, com o objeto de avaliar o efeito do CCL sobre o ganho de peso e as lesões histológicas que ocasiona. Os ratos foram alimentados com carne de coelho, que previamente foi suplementada com CCL. Trinta e cinco dias pós-exposição se registrou o peso corporal; obteve-se a concentração muscular e sérica de CCL através da prova de ELISA, e foram coletados tecidos (fígado e coração) para análise histopatológica. Os valores obtidos dos animais experimentais (Gl e G2) foram analisados mediante um desenho experimental completamente ao acaso com dois tratamentos (n = 10), submetidos a uma análise de variações e comparação de médias com a prova de Tukey (p &lt; 0,05). Registrou-se um aumento de peso corporal de 7 g no G1, contra 4,0 g do G2. O peso do fígado foi de 2,58 g e 1,79, respectivamente (p &lt; 0,05). No G1 a concentração muscular de CCL foi 5324 pg g-1 e em soro sanguíneo de 4378 pg g-1. Somente foram observadas mudanças histológicas em tecidos dos ratos do G1. O fígado apresentou inchaço celular, mitose moderada, picnose e degeneração hidrópica; no coração, engrossamento de fibras, pleomorfismo e filamento nuclear. O CCL favoreceu o aumento de peso nos ratos expostos, e provocou alterações estruturais em fígado e coração.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[clorhidrato de clembuterol]]></kwd>
<kwd lng="es"><![CDATA[ELISA]]></kwd>
<kwd lng="es"><![CDATA[toxicidad]]></kwd>
<kwd lng="es"><![CDATA[ratones]]></kwd>
<kwd lng="en"><![CDATA[clenbuterol hydrochloride]]></kwd>
<kwd lng="en"><![CDATA[toxicity]]></kwd>
<kwd lng="en"><![CDATA[mice]]></kwd>
<kwd lng="en"><![CDATA[ELISA]]></kwd>
<kwd lng="pt"><![CDATA[cloridrato de clembuterol]]></kwd>
<kwd lng="pt"><![CDATA[toxicidade]]></kwd>
<kwd lng="pt"><![CDATA[ratos]]></kwd>
<kwd lng="pt"><![CDATA[ELISA.]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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