<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0123-9392</journal-id>
<journal-title><![CDATA[Infectio]]></journal-title>
<abbrev-journal-title><![CDATA[Infect.]]></abbrev-journal-title>
<issn>0123-9392</issn>
<publisher>
<publisher-name><![CDATA[Asociación Colombiana de Infectología.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0123-93922018000400213</article-id>
<article-id pub-id-type="doi">10.22354/in.v22i4.740</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[The viruses in the human oncogenesis]]></article-title>
<article-title xml:lang="es"><![CDATA[Los virus en la oncogénesis humana]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Vélez-Bohórquez]]></surname>
<given-names><![CDATA[Anggi]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Bohórquez-Lozano]]></surname>
<given-names><![CDATA[Mabel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Echeverry-de-Polanco]]></surname>
<given-names><![CDATA[Magdalena]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad del Tolima Programa de Medicina ]]></institution>
<addr-line><![CDATA[Ibagué ]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad del Tolima Grupo de Citogenética, filogenia y evolución de poblaciones ]]></institution>
<addr-line><![CDATA[Ibagué ]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad del Tolima Grupo de Citogenética, filogenia y evolución de poblaciones ]]></institution>
<addr-line><![CDATA[Ibagué ]]></addr-line>
<country>Colombia</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2018</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2018</year>
</pub-date>
<volume>22</volume>
<numero>4</numero>
<fpage>213</fpage>
<lpage>222</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_arttext&amp;pid=S0123-93922018000400213&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_abstract&amp;pid=S0123-93922018000400213&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_pdf&amp;pid=S0123-93922018000400213&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Based on epidemiological associations and experimentation, relationships between viruses and cancer have been established. For more than 14 million new cases of cancer per year, it is estimated that 15% are related to viral agents. Epithelial, hematolymphoid and mesenchymal malignancies related to different viruses have been document such as Epstein Barr, Kaposi&#8217;s sarcoma, hepatitis B and C, human lymphotropic type 1, Merkel&#8217;s carcinoma and human papilloma. New virus with oncogenic potential such as cytomegalovirus, JC polyoma virus and BK have been described. The interaction of the viruses with the host induces oncogene activation, inhibition of tumor suppressor genes and activation of miRNAs, as determining factors in the development of cancer. The pathology is initiated with the infection that induces the deregulation of cell signaling. The Epstein Barr virus is the oncogenic prototype, with 1% of the human cancers related to it.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen Con base en asociaciones epidemiológicas y experimentación, se ha logrado establecer relaciones entre los virus y el cáncer. Para los más de 14 millones de casos nuevos de cáncer por año, se estima que el 15% se relacionan con agentes virales. Se han documentado malignidades epiteliales, hematolinfoides y mesenquimales, relacionadas con diferentes virus: Epstein Barr, sarcoma de Kaposi, hepatitis B y C, linfotrófico humano tipo 1, carcinoma de Merkel y papiloma humano; se plantean nuevos virus con potencial oncogénico como citomegalovirus, poliomavirus JC y BK. La interacción de los virus con el hospedero muestra activación de oncogenes, inhibición de supresores tumorales y activación de miRNAs, como factores determinantes en el desarrollo de cáncer. La patología se inicia con la infección que induce la desregulación de la señalización celular. El virus de Epstein Barr es el prototipo oncogénico, el 1% de los tipos de cáncer humanos se relacionan con él.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Neoplasms]]></kwd>
<kwd lng="en"><![CDATA[virology]]></kwd>
<kwd lng="en"><![CDATA[pathogenesis]]></kwd>
<kwd lng="es"><![CDATA[Neoplasias]]></kwd>
<kwd lng="es"><![CDATA[virología]]></kwd>
<kwd lng="es"><![CDATA[patogénesis.]]></kwd>
</kwd-group>
</article-meta>
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