<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0123-9392</journal-id>
<journal-title><![CDATA[Infectio]]></journal-title>
<abbrev-journal-title><![CDATA[Infect.]]></abbrev-journal-title>
<issn>0123-9392</issn>
<publisher>
<publisher-name><![CDATA[Asociación Colombiana de Infectología.]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0123-93922020000200081</article-id>
<article-id pub-id-type="doi">10.22354/in.v24i2.837</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Polimorfismos IL1B-511 y TNF-A-308 en una población infectada con Helicobacter pylori de una zona de bajo riesgo de cáncer gástrico en Nariño-Colombia]]></article-title>
<article-title xml:lang="en"><![CDATA[IL1B-511 and TNF-A-308 polymorphisms in a population infected with Helicobacter pylori from a low-risk area of gastric cancer in Nariño-Colombia]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rosero-Rojas]]></surname>
<given-names><![CDATA[Stephany Carolina]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Chaleal-Cultid]]></surname>
<given-names><![CDATA[Javier Antonio]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pazos-Moncayo]]></surname>
<given-names><![CDATA[Álvaro Jairo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rosero-Galindo]]></surname>
<given-names><![CDATA[Carol Yovanna]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Nariño Departamento de Biología ]]></institution>
<addr-line><![CDATA[Nariño ]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Cooperativa de Colombia Facultad de Medicina ]]></institution>
<addr-line><![CDATA[Nariño ]]></addr-line>
<country>Colombia</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>06</month>
<year>2020</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>06</month>
<year>2020</year>
</pub-date>
<volume>24</volume>
<numero>2</numero>
<fpage>81</fpage>
<lpage>87</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_arttext&amp;pid=S0123-93922020000200081&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_abstract&amp;pid=S0123-93922020000200081&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_pdf&amp;pid=S0123-93922020000200081&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Objetivo:  Estudiar los polimorfismos IL1B-511 y TNF-A-308 asociaciados a lesiones precursoras de cáncer gástrico (CG) en una población infectada con Helicobacter pylori (H. pylori) de bajo riesgo de CG de Nariño.  Material y método:  De 105 pacientes con síntomas de dispepsia se incluyeron 81 infectados por H. pylori, (n=63) con gastritis no atrófica y (n=18) con lesiones precursoras de CG de Tumaco: población de bajo riesgo de CG. Las lesiones gástricas se clasificaron por el sistema de Sydney y H. pylori por tinción de Giemsa. Los polimorfismos de IL1B-511 y TNF-A-308 se genotipificaron por PCR-RFLP&#8217;s. Los polimorfismos y su asociación con lesiones gastricas se evaluaron por análisis bivariado y regresión logística binomial.  Resultados: Los pacientes portadores del alelo mutante T (IL-1B-511) no se encontraron a riesgo de lesiones precursoras de malignidad (OR=0,7). No se calculó el OR para TNF-A-308, por fijación del alelo normal G. Ser hombre y estar infectado por H. pylori incrementa 4,3 veces el riesgo de presentar lesiones precursoras de CG y no estar vinculado al régimen de salud aumenta 6,7 veces el riesgo de atrofia gástrica, (OR=4,27 y OR=6,72), respectivamente.  Conclusión:  El alelo mutante T (IL-1B-511) es un biomarcador de resistencia de los pobladores de Tumaco, de bajo riesgo de CG e infectados con H. pylori a padecer lesiones precursoras de CG.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Aim:  To study the IL1B-511 and TNF-A-308 polymorphisms and their possible association with gastric cancer (GC) precursor lesions in a population infected with Helicobacter pylori (H. pylori) of low risk area of GC of Nariño.  Material/method:  105 patients with symptoms of dyspepsia were included, 81 infected with H. pylori, (n=63) with non-atrophic gastritis and (n=18) with precursor lesions of GC of Tumaco: population of low risk of GC. Gastric lesions were classified by the Sydney System an H. pylori by Giemsa staining. The IL1B-511 and TNFA-308 polymorphisms were genotyped by PCR-RFLPs. Polymorphisms and their association with gastric lesions were evaluated by bivariate analysis and binomial logistic regression.  Results:  Patients carryng the mutant T allele (IL-1B-511) were not at risk of precursor lesions of malignancy (OR=0,7). The OR was not calculated for TNF-A-308, by fixing the normal allele G. Being a man and being infected with H. pylori increases 4,3 times the risk of presenting precursor lesions of GC and not being linked to the health regimen increases 6,7 times the risk of gastric atrophy, (OR=4,27 and OR=6,72), respectively.  Conclusion: The mutant T allele (IL-1B-511) is a resistance biomarker of Tumaco residents, low risk of GC and infected with H. pylori to suffer precursor lesions of GC.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Helicobacter pylori]]></kwd>
<kwd lng="es"><![CDATA[Neoplasias estomacales]]></kwd>
<kwd lng="es"><![CDATA[Mutación]]></kwd>
<kwd lng="en"><![CDATA[Helicobacter pylori]]></kwd>
<kwd lng="en"><![CDATA[Stomach Neoplasms]]></kwd>
<kwd lng="en"><![CDATA[mutation]]></kwd>
</kwd-group>
</article-meta>
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