<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1794-3108</journal-id>
<journal-title><![CDATA[Revista Criminalidad]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. Crim.]]></abbrev-journal-title>
<issn>1794-3108</issn>
<publisher>
<publisher-name><![CDATA[Policía Nacional de Colombia]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1794-31082022000300137</article-id>
<article-id pub-id-type="doi">10.47741/17943108.370</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[La neurobiología del asesino: aspectos neuroanatómicos, genéticos, bioquímicos, extrínsecos y sociales]]></article-title>
<article-title xml:lang="en"><![CDATA[The neurobiology of the killer: neuroanatomical, genetic, biochemical, extrinsic and social aspects]]></article-title>
<article-title xml:lang="pt"><![CDATA[A neurobiologia do assassino: aspectos neuroanatômicos, genéticos, bioquímicos, extrínsecos e sociais]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Alemán Ortiz]]></surname>
<given-names><![CDATA[Omar Felipe]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sandoval-Obando]]></surname>
<given-names><![CDATA[Eduardo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Fiscalía General de Justicia de la Ciudad de México  ]]></institution>
<addr-line><![CDATA[Ciudad de México ]]></addr-line>
<country>México</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Autónoma de Chile  ]]></institution>
<addr-line><![CDATA[Santiago de Chile ]]></addr-line>
<country>Chile</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2022</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2022</year>
</pub-date>
<volume>64</volume>
<numero>3</numero>
<fpage>137</fpage>
<lpage>152</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_arttext&amp;pid=S1794-31082022000300137&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_abstract&amp;pid=S1794-31082022000300137&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_pdf&amp;pid=S1794-31082022000300137&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El cuestionamiento del por qué alguien quería asesinar a otra persona aún no tiene una respuesta única: supervivencia, odio, placer, venganza? Frente a este fenómeno, las neurociencias ofrecen un espacio de comprensión relevante y actual para dar respuesta a este interrogante. Diversos estudios clínicos y en neuroimagen han mostrado diversas alteraciones anatómicas, bioquímicas, genéticas y su relación con una variedad de redes neuronales asociadas a conductas agresivas. Subregiones en la corteza prefrontal, la amígdala, el hipocampo y el lóbulo temporal juegan un papel importante en el desarrollo y el funcionamiento de la biología de la agresión. Variaciones genéticas en la regulación de proteínas y neurotransmisores tales como la serotonina y la dopamina han sido establecidas como mediadores en las conductas agresivas y homicidas. Las interacciones epigenéticas y los mediadores sociales representan importantes factores de riesgo adicionales para la agresividad. El presente articulo sistematiza algunos de los factores que influyen en la conducta homicida. Delimita sus factores de riesgo y correlato neurobiológico, así como aporta información basada en evidencia que ayude en la prevención de dichos comportamientos, la comprensión multidimensional del delito y el desarrollo de intervenciones efectivas fundamentadas en las neurociencias cognitivas forenses.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract The question of why someone would want to murder another person still has no single answer: survival, hatred, pleasure, revenge? In the face of this phenomenon, neuroscience offers a relevant and current area of understanding to answer this question. Several clinical and neuroimaging studies have shown diverse anatomical, biochemical and genetic alterations and their relationship with a variety of neural networks associated with aggressive behaviors. Subregions in the prefrontal cortex, amygdala, hippocampus and temporal lobe play an important role in the development and functioning of the biology of aggression. Genetic variations in the regulation of proteins and neurotransmitters such as serotonin and dopamine have been established as mediators of aggressive and homicidal behaviors. Epigenetic interactions and social mediators represent additional important risk factors for aggression. The present article systematizes some of the factors that influence homicidal behavior. It delineates their risk factors and neurobiological correlates, as well as provides evidence-based information to aid in the prevention of such behaviors, the multidimensional understanding of crime, and the development of effective interventions based on forensic cognitive neuroscience.]]></p></abstract>
<abstract abstract-type="short" xml:lang="pt"><p><![CDATA[Resumo A questão de por que alguém quereria matar outra pessoa ainda não tem uma única resposta: sobrevivência, ódio, prazer, vingança? Diante deste fenômeno, as neurociências oferecem uma área relevante e atual de compreensão para responder a esta pergunta. Vários estudos clínicos e de neuroimagem tem mostrado diversas alterações anatômicas, bioquímicas e genéticas e sua relação com uma variedade de redes neurais associadas a comportamentos agressivos. Sub-regiões no córtex pré-frontal, amigdala, hipocampo e lobo temporal desempenham um papel importante no desenvolvimento e funcionamento da biologia da agressão. Variações genéticas na regulação de proteínas e neurotransmissores como a serotonina e a dopamina foram estabelecidas como mediadores de comportamentos agressivos e homicidas. As interações epigenéticas e os mediadores sociais representam fatores de risco adicionais importantes para a agressão. Este artigo sistematiza alguns dos fatores que influenciam o comportamento homicida. Ela delineia seus fatores de risco e correlatos neurobiológicos e fornece informações baseadas em evidências para ajudar na prevenção de tais comportamentos, na compreensão multidimensional do crime e no desenvolvimento de intervenções eficazes baseadas na neurociência cognitiva forense.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Asesinos]]></kwd>
<kwd lng="es"><![CDATA[trastornos del comportamiento]]></kwd>
<kwd lng="es"><![CDATA[neurología forense]]></kwd>
<kwd lng="es"><![CDATA[conducta criminal]]></kwd>
<kwd lng="es"><![CDATA[psicopatología]]></kwd>
<kwd lng="en"><![CDATA[killers]]></kwd>
<kwd lng="en"><![CDATA[behavior disorders]]></kwd>
<kwd lng="en"><![CDATA[forensic neurology]]></kwd>
<kwd lng="en"><![CDATA[psychopathology]]></kwd>
<kwd lng="en"><![CDATA[criminal behavior]]></kwd>
<kwd lng="pt"><![CDATA[Assassinos]]></kwd>
<kwd lng="pt"><![CDATA[distúrbios comportamentais]]></kwd>
<kwd lng="pt"><![CDATA[neurologia forense]]></kwd>
<kwd lng="pt"><![CDATA[comportamento criminoso]]></kwd>
<kwd lng="pt"><![CDATA[psicopatologia]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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