<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2346-3775</journal-id>
<journal-title><![CDATA[Tropical Grasslands-Forrajes Tropicales]]></journal-title>
<abbrev-journal-title><![CDATA[Trop. Grassl.-Forrajes Trop.]]></abbrev-journal-title>
<issn>2346-3775</issn>
<publisher>
<publisher-name><![CDATA[Centro Internacional de Agricultura Tropical (CIAT)]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2346-37752019000200146</article-id>
<article-id pub-id-type="doi">10.17138/tgft(7)146-153</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[An update on leucaena toxicity: Is inoculation with Synergistes jonesii necessary?]]></article-title>
<article-title xml:lang="es"><![CDATA[Una actualización sobre la toxicidad de leucaena: ¿Es necesaria la inoculación con Synergistes jonesii?]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[SHELTON]]></surname>
<given-names><![CDATA[H. MAX]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[KERVEN]]></surname>
<given-names><![CDATA[GRAHAM L.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[DALZELL]]></surname>
<given-names><![CDATA[SCOTT A.]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,The University of Queensland School of Agriculture and Food Sciences ]]></institution>
<addr-line><![CDATA[Brisbane QLD]]></addr-line>
<country>Australia</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Leucaena Research and Consulting Pty Ltd  ]]></institution>
<addr-line><![CDATA[Port Macquarie NSW]]></addr-line>
<country>Australia</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>08</month>
<year>2019</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>08</month>
<year>2019</year>
</pub-date>
<volume>7</volume>
<numero>2</numero>
<fpage>146</fpage>
<lpage>153</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_arttext&amp;pid=S2346-37752019000200146&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_abstract&amp;pid=S2346-37752019000200146&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_pdf&amp;pid=S2346-37752019000200146&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Concern about mimosine toxicity and its management has contributed to the restricted adoption of leucaena as a forage for ruminants. The toxicity is a function of the antimitotic effects of mimosine, which is rapidly converted to isomers of hydroxypyridone (DHP), also toxic compounds, by plant and microbial enzymes. Work by R.J. Jones and colleagues (1960&#8210;1994) identified a rumen bacterium (Synergistes jonesii) capable of degrading DHP, and rumen fluid containing this bacterium was subsequently made available in Australia as a commercial inoculum for cattle producers. Research by University of Queensland and CSIRO over 15 years, commencing in 2003, found evidence for another pathway of toxin management in Indonesia, where hundreds of Balinese farmers had fed uninoculated Bali bulls (Bos javanicus) up to 100% leucaena without experiencing toxicity symptoms, apart from an initial 1&#8210;2 week period while their cattle became adapted to the new diet. Tests showed that the Indonesian cattle were not degrading all DHP, as it appeared in high concentrations in urine samples, predominantly as 2,3-DHP and almost all (&gt;97%) in a conjugated form. The conjugating compounds (glucuronic acid and sulfate compounds), produced in the liver, appeared to be the major pathway for neutralizing the toxicity of DHP. Other work revealed that S. jonesii was a ubiquitous organism in the rumen fluid of animals in all countries but always as a minor population, just detectable using new PCR-based assays, and sometimes not detected in all animals studied. Since the Indonesian cattle fed leucaena suffered symptoms of mimosine toxicity for only a short time before quickly recovering, we hypothesize that conjugation of DHP by the liver was the major detoxification pathway for these animals. This detoxification pathway is also operative in Australia and other countries but further studies are needed to determine its significance.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La preocupación sobre la toxicidad de la mimosina y su manejo ha contribuido a que la adopción de leucaena como forraje para los rumiantes estuviera restringida a nivel mundial. La toxicidad se debe a los efectos antimitóticos de la mimosina, la cual mediante enzimas microbianas y de la planta se convierte rápidamente en compuestos también tóxicos, isómeros de la hidroxipiridona (DHP). Los trabajos de R.J. Jones y sus colegas (1960&#8210;1994) identificaron una bacteria ruminal (Synergistes jonesii) que es capaz de degradar el DHP. Posteriormente el líquido ruminal conteniendo esta bacteria se convirtió en Australia en un inoculante comercial para los productores ganaderos. En investigaciones realizadas por la Universidad de Queensland y CSIRO durante los últimos 15 años se encontró evidencia de otra vía de manejo de toxinas en Indonesia, donde cientos de productores balineses habían alimentado toretes no inoculados de ganado Bali (Bos javanicus) con hasta 100% de leucaena sin experimentar síntomas de toxicidad, aparte de un período inicial de 1&#8210;2 semanas durante el cual los animales se adaptaron a la nueva dieta. Las pruebas mostraron que el ganado indonesio no estaba degradando todo el DHP ya que aparecía en altas concentraciones en muestras de orina, predominantemente como 2,3-DHP y casi todo (&gt;97%) en forma conjugada. Los compuestos de conjugación (ácido glucurónico y compuestos de sulfato), producidos en el hígado, parecieron ser la principal vía para neutralizar la toxicidad del DHP. Otro trabajo reveló que S. jonesii es un organismo ubicuo que se puede detectar en el líquido ruminal de animales en todos los países, pero siempre en poblaciones bajas, muchas veces solo detectables usando nuevos métodos basados en PCR y a veces no detectadas en todos los animales examinados. En vista de que el ganado indonesio alimentado con leucaena mostró síntomas de toxicidad por mimosina solo por poco tiempo y se recuperó rápidamente, nuestra hipótesis es que la conjugación del DHP por el hígado es la principal vía de detoxificación en estos animales. Esta vía de detoxificación también se presenta en Australia y otros países pero se necesitan estudios para determinar su significancia.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Conjugation]]></kwd>
<kwd lng="en"><![CDATA[&#8216;leucaena bug&#8217;]]></kwd>
<kwd lng="en"><![CDATA[microbial detoxification]]></kwd>
<kwd lng="en"><![CDATA[ruminants]]></kwd>
<kwd lng="en"><![CDATA[tree legumes]]></kwd>
<kwd lng="es"><![CDATA[Conjugación]]></kwd>
<kwd lng="es"><![CDATA[detoxificación microbiana]]></kwd>
<kwd lng="es"><![CDATA[leguminosas arbóreas]]></kwd>
<kwd lng="es"><![CDATA[&#8216;leucaena bug&#8217;]]></kwd>
<kwd lng="es"><![CDATA[rumiantes]]></kwd>
</kwd-group>
</article-meta>
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