<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0370-3908</journal-id>
<journal-title><![CDATA[Revista de la Academia Colombiana de Ciencias Exactas, Físicas y Naturales]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. acad. colomb. cienc. exact. fis. nat.]]></abbrev-journal-title>
<issn>0370-3908</issn>
<publisher>
<publisher-name><![CDATA[Academia Colombiana de Ciencias Exactas, Físicas y Naturales]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0370-39082019000300351</article-id>
<article-id pub-id-type="doi">10.18257/raccefyn.792</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Filogenia y oncogénesis del virus del papiloma humano: una aproximación translacional al descubrimiento de biomarcadores para la detección de lesiones precancerosas de cérvix]]></article-title>
<article-title xml:lang="en"><![CDATA[Phylogeny and oncogenicity of the human papillomavirus: A translational approach for the discovery of biomarkers for the detection of precancerous lesions of the cervix]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Ramírez-Pineda]]></surname>
<given-names><![CDATA[Arianis Tatiana]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[González]]></surname>
<given-names><![CDATA[Martha Isabel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Castañeda-Vanegas]]></surname>
<given-names><![CDATA[Kelly Melisa]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Agudelo-Fernández]]></surname>
<given-names><![CDATA[María Cecilia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[López-Urán]]></surname>
<given-names><![CDATA[Carolina]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Sánchez-Vásquez]]></surname>
<given-names><![CDATA[Gloria Inés]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Antioquia Facultad de Medicina Grupo de Infección y Cáncer]]></institution>
<addr-line><![CDATA[Medellín ]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad de Antioquia Facultad de Medicina Departamento de Patología]]></institution>
<addr-line><![CDATA[Medellín ]]></addr-line>
<country>Colombia</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>09</month>
<year>2019</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>09</month>
<year>2019</year>
</pub-date>
<volume>43</volume>
<numero>168</numero>
<fpage>351</fpage>
<lpage>365</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_arttext&amp;pid=S0370-39082019000300351&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_abstract&amp;pid=S0370-39082019000300351&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.co/scielo.php?script=sci_pdf&amp;pid=S0370-39082019000300351&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El cáncer cervical es la principal causa de mortalidad entre las mujeres en poblaciones con índice de desarrollo humano bajo y medio. La infección con 14 de los genotipos del virus del papiloma humano de alto riesgo (VPH-AR) es la causa principal de este cáncer. Las vacunas profilácticas previenen esta infección si se aplican antes del inicio de la actividad sexual. Las mujeres que no han sido vacunadas deben someterse a tamización con la prueba de VPH-AR, y recibir el diagnóstico histopatológico y el tratamiento oportunos. Cerca del 20 % de las mujeres son positivas para el VPH-AR, pero muy pocas presentan lesiones potencialmente malignas. Las mujeres infectadas con los genotipos VPH16 o 18 o aquellas con ascendencia europea infectadas con variantes de origen nativo americano del VPH 16 tienen mayor riesgo de cáncer. También aquellas con historia de uso de anticonceptivos hormonales y de tabaquismo. Estos factores parecen contribuir a un aumento de la expresión de las oncoproteínas E6 y E7 del VPH-AR, lo que conduce a la desregulación del control del ciclo celular y, finalmente, al cáncer. En el presente estudio se pasa revista al estado del arte de los mecanismos carcinogénicos del VPH-AR haciendo énfasis en el papel de las proteínas E6 y E7 y se presentan los resultados de la genotipificación del VPH16 o 18, la tasa de positividad de las proteínas p16 y ki67 y la evaluación bioinformática de la expresión aberrante de los miARN y su correlación con el grado de la lesión cervicouterina, datos que contribuyen a validar el potencial clínico de estos biomarcadores para la detección de lesiones potencialmente malignas de cérvix.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Cervical cancer is the main cause of mortality among women in populations with low and medium human development index. The infection with 14 genotypes of the high-risk human papillomavirus (HR-HPV) is the main cause of this cancer. Prophylactic vaccines prevent this infection if they are applied before the onset of sexual activity. Unvaccinated women should be screened with the HR-HPV test and receive the histopathological diagnosis and timely treatment. About 20% of women are HR-HPV positive, but very few have potentially malignant lesions. Women infected with HPV16 or 18 genotypes or women with European ancestry infected with Native American variants of HPV 16 have a higher risk of cancer. Also, those with a history of use of hormonal contraceptives and cigarette smoking. These factors seem to contribute to an increase in the expression of the HR-HPV oncoproteins E6 and E7, which leads to the deregulation of cell cycle control and finally to cancer. We present here the state of the art of the carcinogenic mechanisms of HPV-AR emphasizing the role of the E6 and E7 proteins, as well as the results of our study on the HPV16 or 18 genotyping, the positivity rate of the p16 and ki67 proteins, and the bioinformatic evaluation of the aberrant expression of miRNAs and their correlation with the degree of cervical lesions. These data contribute to validate the clinical potential of these biomarkers for the detection of potentially malignant lesions of the cervix.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Virus del papiloma humano]]></kwd>
<kwd lng="es"><![CDATA[Variantes virales]]></kwd>
<kwd lng="es"><![CDATA[Filogenética]]></kwd>
<kwd lng="es"><![CDATA[Cáncer cervical]]></kwd>
<kwd lng="es"><![CDATA[Biomarcadores]]></kwd>
<kwd lng="es"><![CDATA[Prevención secundaria]]></kwd>
<kwd lng="en"><![CDATA[Human papillomavirus]]></kwd>
<kwd lng="en"><![CDATA[Viral variants]]></kwd>
<kwd lng="en"><![CDATA[Phylogenetic]]></kwd>
<kwd lng="en"><![CDATA[Cervical cancer]]></kwd>
<kwd lng="en"><![CDATA[Biomarkers]]></kwd>
<kwd lng="en"><![CDATA[Secondary prevention]]></kwd>
</kwd-group>
</article-meta>
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